The Cell-Symbiosis Therapy following Dr. Kremer

Ketogenic Nutrition following Dr. Coy


The Cell-Symbiosis Therapy following Dr. Kremer
In the countries of the western world one person in 3 develops some form of cancer, and every 4th tumour patient dies of it. According to forecasts made by the WHO, by the year 2050 every 2nd death will be caused by cancer.

NAccording to the dominating cancer theory, the primary cause of the illness is held to be incidental defects (mutations) in the DNA of the cell nuclei that are seen as being irreparable. This is the assumption on which the standard treatments in cancer medicine (operation, chemotherapy and/or radiotherapy) are based. The healing rates for cancer (= survival of at least 5 years after the diagnosis) are currently put at 45% (22% from surgical treatment, 12% from radiotherapy, 5% from chemotherapy, and 6% from a combination of the standard therapies).

In the treatment of incurable cancers, the so-called palliative treatment, some 60-70% of the patients are treated with radiotherapy, 50% with chemotherapy and less than 1% by surgery (EU data, 2003). In den USA, for example, 20% of the total budget expended on public health each year is used for chemotherapy of patients with cancer.

The Nobel Prize winner Prof. Watson, who together with Crick discovered the structure of the so-called double helix of DNA in the cell nucleus and is the most prominent promoter of the "War on Cancer" campaign initiated in the USA in 1971, stated concisely in 2002: "We first have to understand cancer, before we can heal it."

The background to this sobering summary, after decades of the most intensive research efforts with enormous capital investment, is the fact that the classical mutation theory in cancer genesis has been lastingly devastated by more recent research findings.

According to the mutation theory a tumour colony arises from a single "degenerate" body cell which, by means of uncontrolled division of the DNA-defect identical in each case, passes this on to each daughter cell. He had discovered, however, that each single cancer cell even within the same tumour of a patient had its own distinctive genetic variation.

The international acclaimed cancer researcher Prof. Weinberg from the MIT in Cambridge (USA) and Prof. Hahn of the Dana Farber Cancer Research Center in Boston, both adherents of the classical mutation theory of cancer genesis, published (in 2002) an overview of the still-puzzling "6 Devilish Properties of Cancer Cells":

1. Cell division, even without external growth signals
2. Growth despite stop signals from the neighbouring cells
3. Avoidance of the self-destruction program
4. Stimulation of the new formation of blood vessels
5. Acquisition of potential immortality
6. Invasion and formation of metastatic daughter cells.

The cell-symbiosis concept (Kremer 2001) explained for the first time the so called "Devilish Properties" of the cancer cells as an evolutionary biological programming of the natural (though overdriven) protection of the division activities of human cells under circumstances of over-excessive chronic stress on the cells. The starting point of this concept is the evolutionary biological discovery that mankind owes its biological existence – as do all cell-nucleus-containing one-cell and multi-cell living organisms (eukaryotes) – to a unique act of combination in evolutionary history. About 1.5 --2 billion years ago two single cells from the spheres of the archaea and the bacteria, each without a cell nucleus, merged to form a new type of single cell, nowadays known as protists. Extensive comparisons of sequential analyses of the genotype and certain proteins between archaea, bacteria and a large number of eukaryotic organisms including mankind gave a surprising result.

Some 60% of the genes in the human cell nucleus stem from the original archaeon (A genomes), whereas the remaining genes are of bacterial origin (B genomes), especially in the cell nuclei delegated from the bacterial fusion partners that have survived in all human cells as the so-called mitochondria (average number per cell: 1500).

The A genome and the B genome practice controlled division of duties.

The A genome dominates the later cell-division phases, the B genome controls the early cell-division phase and the differentiated cell performances of the respective types of cells in the tissue association.

From this fundamental cell-biological fact, the cell-symbiosis concept – on the basis of a large amount of new and integrated experimental and clinical research data – arrives at the following conclusions regarding cancer genesis and cancer therapy:

1. There is a controlled behavioural interchange between the mitochondria and the two cell-nuclei sub-genomes.

2. In the event of transformation to cancer cells a functional (not a structural) disturbance to this behavioural interchange occurs. The cells, after the cell-division phase, can no longer adequately switch back to discharge their differentiated cell-performance phases.

3. The cause for this enduring malfunction is the gradual failure of one of the central functions of the mitochondria. They provide, for nearly all of the biosynthetic and metabolic processes, approx. 90% of the "universal energy-storage and energy-transfer molecules", adenosine phosphate (ATP). For normal requirements, an amount of ATP roughly equivalent to the weight of one's body must be synthesized every day and then decomposed again. ATP cannot be stored. The natural ATP reserve for the human organism is adequate for only about 5 seconds.

In the event of impaired functioning of the mitochondria, cancer cells switch essentially to the archaic oxygen-dependent form of ATP synthesis in the cell plasma, with a glucose turnover increased by a factor of up to 20, at the cost of the entire organism (emaciation of the cancer patient being one of the main causes of death), which in the case of an intact immune system, during the phase preceding that of clinical manifestation, can be demonstrated by the tumour marker TKTL1 in the macrophages.

4. The previous concepts relating to the synthesis and functioning of the ATP molecule, the basis of all cell-biological medical theories, are however objectively incorrect.

ATP has three molecular groups: 1 alkaline adenine group, that absorbs light quantums maximally near to the ultraviolet area of 270 nm; 1 sugar molecule with 5 carbon atoms; and 1 molecule tail with 3 phosphate groups.

The valid dogma to date is the theory propounded by the later Nobel Prize winner Lippmann, that in the so-called respiratory chains of the mitochondria – of which there are thousands in each mitochondrion, as shown by electron-microscopic photos – the current of "high-energy" electrons from the nutritious substances via a sort of electrochemical battery transfer their electron energy to protons. These are then said, for their part, to drive the ATP synthesis and to store their excessive energy in the phosphate bonds of the ATP. By water-splitting of the "high-energy" phosphate bonds of the ATP transported in the cell plasma, all of the energy processes of cell metabolism are maintained.

Biochemical investigations, on the other hand, have clearly shown that the phosphate bonds of the ATP are by no means especially energetic and in the course of water-splitting would only release enough heat energy for use by isothermic cells (i.e. steady temperature) and at best for heat production.

The fundamental questions as to the actual mechanism employed for production of cell energy are therefore fully open. This fact explains the relative failure of the previous attempts at cancer prevention and therapy.

5. The fields of biochemistry and medicine have also been unable so far to explain the function of the adenine group of the ATP, since no biochemical reaction with this molecule has been able to be demonstrated. Such an understanding is given, however, in the context of the cell-symbiosis concept from the biophysical characteristic of light absorption by the adenine group. All significant components of cell respiration in the mitochondria are light-absorbing molecules with characteristic "frequency windows" of the absorption maximum of the near UV area to the long-wave yellow-orange-coloured spectral area of visible light at approx. 600 nm.

The source of the electromagnetic energy is not, however, sunlight. Instead it is the steady flow of unpaired, paramagnetically-aligned electrons in the respiratory organs that induce a low-frequency, pulsing, electromagnetic field. The electromotor force produced in this way is magnified enormously, in a catalytic manner, by the enzyme complexes of the respiratory chains and effects the interaction between the electrons and the protons, which are also paramagnetically aligned. This process creates a quantum-dynamic information transfer in the form of photon-exchange energy. The photons, as a source, are fluctuations of resonance frequency in the physical vacuum (zero-point energy field). The information transferred is stored in the angular momentum (spin) of the protons themselves, which for their part achieve an ATP synthesis complex by way of a proton gradient. Here, the resonance information is transferred to the adenine group of the ATP by a unique rotation system, the electrons of the group being freely mobile in an alternating double bond of the ring molecule. The ATP serves, in other words, as an "antenna molecule" for receipt and passing on of resonance information from the "morphogenetic background field". Human cell symbiosis is thus no heat engine, but a light-frequency-modulated, information-conversion media.

6. At the time of cancer genesis the 4th enzyme complex of the respiratory chains in particular is, for a variety of reasons, functionally disturbed. In accordance with the conventional concept, this complex performs the duty, at the end of the respiratory chains, of transferring the inflow of electrons to the molecular oxygen, reducing this in the process to the water molecule (H2O).

The decisive factor from the standpoint of the cell-symbiosis concept, however, is the electron pairing achieved during the reduction (deoxygenation) of O2 to water, this generating an antimagnetic impulse that switches the electromagnetic alternating field for resonance information transfer on and off at a very fast rate of time (pico-seconds). If the electron flow to the 02 molecule is enduringly disturbed, however, a faulty modulation of the ATP will take place and oxygen radicals and other radicals will form and accumulate, these being able to attack and damage the macromolecules (nucleic acids, proteins, lipids, carbohydrates).

To avoid this danger, the key enzyme haeme oxygenase is massively upgraded. This enzyme makes use of 02 as a cofactor for the production of carbon monoxide (CO). In the event of enduring excessive production the CO gas exerts decisive effects on cancer-cell transformation:

- CO gas effects a characteristic phase displacement of the absorption of the visible light by components of the respiratory chains and consequently, so to speak, a short circuiting in the photon switcher for the modulation of the information transfer to the mitochondrial ATP.

- CO gas activates certain regulatory proteins in the cell plasma (cytoplasm) for stimulation of the cell-division cycle even without external growth signals (1. "Devilish Properties of Cancer Cells", see above)

- CO gas effects, via enzymatic over-activity of the important secondary messenger cyclic guanosine monophosphate (c GMP), the inhibition or blockage of the communication between the cells in the tissue association (2. "Devilish Properties of Cancer Cells")

- CO gas blocks the "programmed cell death" by bonding to the bivalent iron in important key enzymes (3. "Devilish Properties of Cancer Cells").

7. According to more recent clinical findings, cancer cells develop particular "malignance" and massively spread metastatic cells if the 02 supply to the tumour cells by the capillary blood vessels is blocked. In such cases chemotherapy and radiotherapy are no longer effective, since without the presence of molecular oxygen the programmed death of the cancer cells can no longer be induced. Cancer patients in this situation are declared by school medicine as being "out-therapied".

The cell-symbiosis concept postulates that, if the cofactor O2 is missing, instead of CO gas the much more effective cyanide (CN) gas will be formed. In connection with humans CN is regarded as the strongest mitochondrial respiratory poison and presumably gives rise – via the known reduction inhibition of the trivalent iron of the haeme cytochrome of the respiratory chains to the bivalent iron – to a still stronger phase displacement of the absorption of visible light. This hypothesis can be said to support the evolution-biological standpoint of the cell-symbiosis concept, that cancer cells effectively revert back to "protozoons" (due to the loss of cell-to-cell communication with the neighbouring tissue cells) and therefore behave like "cell parasites" (4., 5. and 6.: "Devilish Properties of Cancer Cells"). In this sense cancer cells represent a regression to the earlier eukaryotic stage of the monocellular protists, making use in the process, as a survival strategy, of the human evolution archives preserved in the cell genomes, to adapt to the existing milieu conditions faced by the individual cancer cell (individual genetic variation, see above).

In 2003 the functional disturbance of cancer cells in the 4th complex of the respiratory chains with simultaneously intact messenger RNA and intact mitochondria DNA was confirmed by US cancer researchers, without the specialists being able to explain the phenomena.

8. At the end of 2002, however, a cancer-research group at the University of Helsinki, after many years of animal experiment and clinical studies, were able for the first time to document exactly, using the electron microscope and mass spectrometry, that the transformation to cancer cells is indeed caused by loss of control of the mitochondria over the cell-division cycle. By means of a specific, experimentally determined, biological compensation therapy the clinical research team was able to demonstrate in various cases of human cancerous illness that the tumour cells, after a relatively short time, had reprogrammed themselves to intact, normal, differentiated cells, whereas no programmed cell death was verifiable.

In 2003 researchers at the Andersan Cancer Research Center of the University of Texas in Houston published the first comprehensive review of hundreds of animal-experimental studies on the effects of curcuma – the component of turmeric (Curcuma longa. of the ginger family of plants) – on cancer cells and metastases. The researchers were astonished to find that curcuma effectively blocked all signal paths in tumour cells and metastases. The researchers were unable to give an explanation for this broadband effect.

The effects induced by curcuma can however be explained when one knows that curcuma is highly absorbent in the violet range of the spectrum of visible light. It has exactly the same wavelength, at 415 nm, as the electron transferring molecule cytochrome c, which is forcedly decomposed in cancer cells by the protective enzyme haeme oxygenase. Curcuma therefore bridges, so to speak, the short circuit in cancer cells in the photon switcher between the 3rd and 4th complex of the respiratory chains in the mitochondria and thereby normalizes the information transfer for the correct functional modulation of the ATP.

The concept of the cell-symbiosis therapy (Krämer 2001), derived from the findings of cell-symbiosis research, has meanwhile led to spectacular therapeutic successes – even in individual cases of cancerous illness declared by school medicine to be incurable. A broad spectrum of naturally light-active (classes of) substances is available. The potential is by no means as yet exhausted.

What is desperately needed, however, is a comprehensive review of the current status of research with the aim of developing optimal therapeutic recipes and making these available for practical clinical and therapeutic use. To this end – as an undertaking for an interdisciplinary research group – it is not however to be expected that research sponsorship will be provided by the established health system in the foreseeable future.

Following Dr. med. Kremer



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Ketogenic nutrition following Dr. Coy
Some tumours are characterized by invasive growth and the formation of metastases. They acquire their energy from the fermentation of glucose, even if sufficient oxygen for combustion is present. Thanks to the discovery of the enzyme TKTL1 this phenomenon can now be biochemically explained for the first time and enables new therapeutic approaches. In TKTL1-positive cells the burning of fat (beta-oxidation) is switched off. This is why these cells are dependent on glucose as a source of energy. It is on the basis of this knowledge that the TKTL1 nutrition therapy was developed.

TKTL1 Nutrition Therapy
TAVARLIN® Products Enable Implementation of a TKTL1 Nutrition Therapy.

This is a therapy aimed at the metabolism of aggressive, fermenting cancer cells. The aim is to separate the cancer cells from their energy supply, so as to be able to influence their growth and cell division.

Das TAVARLIN® Concept makes use of various foodstuffs that discharge a variety of functions.

a) They have an inhibitory effect on TKTL1 metabolism.

b) They secure the organism's energy supply. Since for the TKTL1 nutrition therapy energy sources such as sugar and carbohydrates have to be avoided, the missing calories must be provided in the form of other nutritious substances, such as high-quality oils and protein.

c) They ease implementation of the TKTL1 nutrition therapy. This is why products like TAVARLIN® Protein-Nudeln and TAVARLIN® Protein-Brot und Kuchen, which are specially made of protein, are included in the nutritional package.

Following Dr. Coy


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